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Calutide (Bicalutamide)
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Bicalutamide

Calutide is a widely used, relatively non-toxic anti-androgen, particularly when used in combination with androgen deprivation. In men on combined androgen blockade (CAB), the typical dose is 50 mg per day. For men receiving monotherapy with Calutide anti-androgen, the dose is 150 mg per day. The objective was to determine the PSA response rate to increasing Calutide to 150 mg per day in men who develop castrate-resistant prostate cancer (CRPC) on CAB with goserelin acetate and Calutide 50 mg per day.

Other names for this medication:
Bicalutamide, Casodex, Bicalox, Bicalutamida, Bicusan, Bidrostat, Caluran, Calutide, Calutol, Capro, Cosudex, Imda, Lutamidal, Probic, Zolacos,

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Calutide is used together with a luteinizing hormone releasing hormone (LHRH) analog (eg, goserelin or leuprolide) to treat stage D2 metastatic prostate cancer (cancer that has spread) in men. Calutide belongs to the group of medicines called antiandrogens. It works by blocking the effects of testosterone (a male hormone), which helps stop the growth and spread of cancer cells.

This medicine is available only with your doctor's prescription.

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MicroRNAs as Circulating Biomarkers of Left Ventricular Remodeling after Myocardial Infarction. There was no evidence that women who had a cesarean delivery experienced fewer sexual health problems. The mechanism that provides the basis for its cardiovascular stability is the capacity to bind and stimulate peripheral alpha-2B adrenergic receptors with a subsequent vasoconstriction. CAV-1 and CAV-2 are expressed in normal and glaucoma tissue and TM cell lines. Clinical success was more common in FN patients randomized to meropenem compared with the patients randomized to benzylpenicillin plus an aminoglycoside.

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Most X-linked microRNA genes escape silencing and are expressed in testis. Preventing sleep on the first resting phase following a traumatic event attenuates anxiety-related responses. Hypertriglyceridemia and smoking are determinants of LOX-1 ligand activity in healthy men and are thus thought to be crucial risk factors for initiation of atherosclerotic progression through generation of OxLDL. Fluorescent microscope analysis showed that HepG2 cells internalized Cur-NLC more effectively than native curcumin.

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